What Is The Difference Between Hash And Marijuana?

What Is The Difference Between Hash And Marijuana
The Distinction Between Hash and Cannabis – First, “what is the difference between a hash and marijuana high?” is an often asked question. Since there are several forms of cannabis that may be cultivated, the duration of a high will depend on the strain.

  1. People who acquire hash must be cautious, as the material frequently contains fillers such as sand, plastic, or even excrement.
  2. Hashish is a condensed version of the cannabis plant, therefore its cannabinoid content is high or strong.
  3. Unlike hash, marijuana is derived from the cannabis plant’s dried leaves, seeds, stems, and flowers.

The hue is distinct from that of hash, since it is often a tint of green. Marijuana, or “Mary Jane,” is utilized for a variety of reasons, including medical treatment, the creation of edibles, and inclusion in teas. Both hash and marijuana can be smoked by rolling a joint or blunt, inhaling the drugs via a bong or pipe, or by smoking “dabs,” the concentrated oil, through a rig.

Which is more potent: cannabis or skunk?

The Mail on Sunday has proclaimed that cannabis “TRIPLES the chance of psychosis” and is responsible for “1 in 4 new major mental diseases.” Is this what the cited study indicates? No, but it’s extremely fascinating research that might help us better understand the association between cannabis and psychosis.

The research contrasts a sample of psychosis patients with their general community counterparts in South London. The authors asked individuals not only about the frequency of their cannabis usage, but also about the sort of cannabis they consumed, namely skunk or hash. They discovered that psychosis patients were much more likely to have used skunk daily than to have never used cannabis.

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In contrast, daily hash smokers were no more likely to develop psychosis than those who had never tried cannabis. But does this suggest that cannabis triples the risk of psychosis? The Mail’s headline was derived from a ‘population attributable percentage’ that the publication calculated: the number of psychosis cases that would be averted if all cannabis usage was eliminated, presuming cannabis causes psychosis.

This was assessed by the study’s authors to be 24%. However, the authors emphasize that they cannot be certain that the observed connection is causative. There is substantial evidence linking cannabis usage to psychosis and schizophrenia, but as the phrase goes, correlation does not imply causality. You cannot arbitrarily designate one set of teens to use cannabis while another group does not.

This necessitates observing what individuals choose to do, and those who choose to use cannabis may differ in a number of other ways, which might be the source of the increased psychosis risk. Although you can control for them in analyses, you can never be certain that you have done so properly.

This study used a case-control methodology. This sort of study might be biased if the control population is not properly chosen. This article’s controls consisted of London residents from the same neighborhood. In particular, individuals with psychosis were more likely to be male, white, and heavy smokers than those in the control group.

Psychosis is more prevalent among men and ethnic minorities (in particular those of migrant status). The authors accounted for these differences and other potential confounding variables, but there may have been more that were not examined. Another disadvantage of case-control studies is that the exposure measure (cannabis usage) must be retrospectively assessed.

This might lead to skewed results if psychosis influences the risk of over- or under-reporting previous drug usage. Given that a correlation was observed with skunk but not hash, it is possible that this issue is improbable, but the authors cannot be certain. Despite the drawbacks, this study is extremely significant.

It is the first attempt of its sort to isolate cannabis potencies in this manner. Past cannabis research has mostly focused on the effects of THC, which has been proven to elicit brief psychotic episodes in randomized studies. However, recent data suggests that another cannabinoid, cannabidiol (CBD), may be protective against psychosis.

  • Skunk often includes greater quantities of THC than hash (government reports indicate 15% in skunk and 5% in hash), however skunk typically contains only trace amounts of CBD, whereas hash typically has CBD and THC levels that are about similar.
  • In this study, skunk and hash usage was self-reported, so we must presume that participants understood what they were smoking and can only speculate about the actual THC and CBD amounts in the cannabis consumed.
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In a perfect world, samples would have been collected and the exact quantities of these cannabinoids would have been determined, however this is impractical for research of this scale and design. If it is true that skunk usage increases the chance of psychosis, does this indicate that it is responsible for one in four major mental problems, as claimed by the Mail? Ignoring the fact that this paper was specifically about psychosis and not’serious mental disorders’ in general, the impact of cannabis may be this great in certain high-risk populations, such as those with a genetic predisposition to psychosis, but it’s a huge leap to draw such a conclusion from a single study.

This new study is an essential step toward understanding the nature of the relationship between cannabis and psychosis, but once again, media exaggeration or misinterpretation of the findings might jeopardize the message’s ability to reach those who would most benefit from it. Similar to how a pint of beer in the evening is likely to have a different effect on health than a pint of vodka, skunk may have a different effect on health than hash.

I anticipate more investigation into this topic. At the time I wrote this, only the Mail had reported on the study. However, the Guardian also published the title “skunk triples the chance of a catastrophic psychotic episode” while the Telegraph said that “super-potent cannabis is responsible for a quarter of all new instances of psychosis.” So please accept our apologies for singling out the Mail! Suzi Gage has just finished her doctoral dissertation on the relationship between cannabis usage and psychotic episodes among Bristol’s Children of the 1990s cohort, and has published both a review and her results on the subject.

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